🎙️ Podcast Segment: Amyloidosis — PYQ Breakdown & Concept Mastery
❓ PYQ Example
Question: Which stain is used to identify amyloid deposits in tissue sections?
A. PAS
B. Congo red
C. Hematoxylin
D. Ziehl-Neelsen
✅ Correct Answer: B. Congo red
---
🧠 Concept Breakdown
- Amyloidosis is a condition where misfolded proteins deposit extracellularly in tissues.
- These proteins form β-pleated sheets, giving them unique staining and birefringence properties.
- Types of Amyloid:
- AL (light chain) — seen in multiple myeloma
- AA (acute phase reactant) — seen in chronic inflammation
- Aβ — seen in Alzheimer’s disease
- Staining: Congo red stain shows apple-green birefringence under polarized light — a classic PYQ clue.
---
⚠️ Caution for Students
- Don’t confuse PAS stain (used for glycogen) with Congo red for amyloid.
- Know the clinical associations — e.g., nephrotic syndrome, restrictive cardiomyopathy, macroglossia.
- Amyloid deposits are extracellular, not intracellular — a common MCQ trap.
---
🩺 Clinical Relevance
- Systemic amyloidosis affects kidneys, heart, liver, and nerves.
- Localized amyloidosis includes Alzheimer’s and medullary thyroid carcinoma.
- Diagnosis involves biopsy + Congo red staining + clinical correlation.
---
🎙️ Title
"Amyloidosis Uncovered: PYQ Mastery for NEET PG & FMGE"
📝 Description
This episode tackles one of the most conceptually tricky and frequently asked topics in NEET PG/FMGE/INICET — Amyloidosis. Learn the types, staining techniques, clinical relevance, and how to avoid common traps in under 10 minutes. Designed by a medico, for medicos.
NEETPG #FMGEprep #INICET #Amyloidosis #PathologyPYQ #MedicoPodcast #RapidReview #MedicalStudents #ExamPrep #MBBS #HighYieldPathology #SpotifyPodcast
`
Podcast Segment: Inflammation — PYQ Breakdown & Concept Mastery
❓ PYQ Example
Question: Which of the following is a cardinal sign of acute inflammation?
A. Pallor
B. Tumor
C. Cyanosis
D. Atrophy
✅ Correct Answer: B. Tumor (Swelling)
---
🧠 Concept Breakdown
- Acute inflammation is the body’s rapid response to injury or infection.
- Cardinal signs (Celsus + Virchow):
- Rubor (Redness)
- Calor (Heat)
- Tumor (Swelling)
- Dolor (Pain)
- Functio laesa (Loss of function)
- Mediators: Histamine, prostaglandins, bradykinin, cytokines
- Vascular changes: Vasodilation, increased permeability, leukocyte migration
---
⚠️ Caution for Students
- Don’t confuse tumor (swelling) with neoplastic tumor — context matters!
- Pallor and cyanosis are not signs of inflammation — common distractors.
- Know the sequence of events: vasodilation → permeability → exudation → neutrophil migration
---
🩺 Clinical Relevance
- Seen in infections, trauma, autoimmune conditions
- Understanding mediators helps in pharmacology (NSAIDs, steroids)
- Chronic inflammation leads to fibrosis, granulomas, and tissue damage
---
🎙️ Title
"Inflammation Demystified: PYQ Mastery for NEET PG & FMGE"
📝 Description
This episode breaks down a classic PYQ on inflammation — one of the most tested topics in Pathology. Learn the cardinal signs, key mediators, and clinical relevance in under 10 minutes. Designed by a medico, for medicos.
NEETPG #FMGEprep #INICET #PathologyPYQ #InflammationRevision #MedicoPodcast #RapidReview #MedicalStudents #ExamPrep #MBBS #HighYieldPathology #SpotifyPodcast
This episode breaks down a classic PYQ on apoptosis vs necrosis — a must-know concept for NEET PG, FMGE, and INICET aspirants. Learn the differences, avoid common traps, and understand the clinical relevance in under 10 minutes. Designed by a medico, for medicos.
Apoptosis vs Necrosis — PYQ Breakdown
🧪 PYQ Question
Which of the following is a feature of apoptosis but not necrosis?
A. Cell swelling
B. Inflammatory response
C. DNA fragmentation
D. Loss of membrane integrity
✅ Correct Answer: C. DNA fragmentation
---
🧠 Concept Breakdown
- Apoptosis is a programmed, energy-dependent cell death — clean and non-inflammatory.
- Necrosis is accidental, uncontrolled cell death — messy and inflammatory.
- Key differences:
- Apoptosis: Cell shrinkage, chromatin condensation, DNA fragmentation, intact membrane.
- Necrosis: Cell swelling, membrane rupture, inflammation.
---
⚠️ Caution for Students
- Don’t confuse cell swelling (necrosis) with cell shrinkage (apoptosis).
- Inflammation is absent in apoptosis — a common MCQ trap.
- DNA laddering is a lab clue for apoptosis.
- Image-based questions often show apoptotic bodies — small, round fragments.
---
🩺 Clinical Relevance
- Apoptosis: Seen in embryogenesis, immune regulation, cancer therapy.
- Necrosis: Seen in ischemia, infections, toxins — e.g., myocardial infarction.
#NEETPG #FMGEprep #INICET #PathologyPYQ #ApoptosisVsNecrosis #MedicoPodcast #RapidReview #MedicalStudents #ExamPrep #MBBS #HighYieldPathology #SpotifyPodcast
From cell injury to neoplasia, this episode delivers a punchy review of essential pathology topics for NEET PG, FMGE, and INICET aspirants. Designed for medicos on the move, we cover high-yield facts, buzzwords, and clinical pearls — all in a format that’s easy to absorb and hard to forget.
Whether you're revising late-night or squeezing in a quick listen during rounds, this episode helps you stay exam-ready and confident.
#PathologyRevision #NEETPG #FMGEprep #INICET #MedicoPodcast #MedicalStudents #HighYieldPathology #ExamPrep #MBBS #MedSchoolTips #SpotifyPodcast #RapidReview #StudyWithMe
Struggling to retain anatomy before your NEET PG, FMGE, or INICET exam? This crisp 10-minute revision episode is your go-to booster. We cover high-yield topics, quick mnemonics, and clinical correlations that matter — all in a format designed by a medico, for medicos.
Whether you're commuting, cramming, or casually brushing up, this episode helps you stay sharp and exam-ready. Tune in, revise smart, and ace your prep!
NEETPG #FMGEprep #INICET #MedStudentLife #AnatomyRevision #MedicoPodcast #RapidReview #ExamPrep #MBBS #MedicalPodcast #SpotifyPodcast #StudyWithMe #MedSchoolTips #HighYield
🎙 Dr. StemCell Podcast
Episode Title: Acute Pancreatitis — High-Yield NEET PG Quick Shot
---
📘 Podcast Description:
In this high-yield NEET PG episode, Dr. StemCell breaks down Acute Pancreatitis—a classic PYQ magnet. From scoring systems to enzyme markers, this 4-minute blitz gives you everything you need to ace the topic. Learn how to spot the clinical clues, avoid common traps, and master the treatment algorithm.
What You’ll Learn:
- Most common cause: Gallstones > Alcohol
- Key diagnostic markers: Serum lipase > amylase
- Scoring systems: Ranson’s, BISAP
- Imaging: CT scan after 72 hrs if diagnosis unclear
- Treatment: Supportive care, fluids, NPO, pain control
Mnemonic Mastery:
“GET SMASHED” for causes
“PANCREAS” for Ranson’s criteria
🎯 NEET PG Motto:
"Every PYQ is a blueprint. This podcast turns past questions into future victories."
#NEETPG #NEETPG2026 #NEETPGPreparation #NEETPGExam #NEETPGSyllabus #NEETPGStudyPlan #NEETPGCoaching #NEETPGTopperTips #HowToCrackNEETPG #NEETPGQuestionPaper #NEETPGMockTest #NEETPGStrategy #NEETPGTips #NEETPGPreviousYearQuestions #NEETPGOnlineCoaching #NEETPGDailyRoutine #NEETPGNotes #NEETPGImportantTopics #NEETPGStudyMaterial #NEETPGExamPattern #BestBooksForNEETPG #MBBS #MedicalStudents #MedicalEducation #MedSchool #MedStudy #Anatomy #Physiology #Biochemistry #Pathology #Pharmacology #Microbiology #Medicine #Surgery #OBGYN #Pediatrics #Radiology #Orthopedics #Dermatology #Psychiatry #Anesthesia #ENT #Ophthalmology #EmergencyMedicine #CommunityMedicine #INICET #FMGE #AIIMS #JIPMER #PGEntrance #MedicalEntrance
Dr. StemCell Podcast
Episode Title: Hyponatremia — High-Yield NEET PG Quick Shot
---
📘 Podcast Description:
In this crisp 4-minute episode, Dr. StemCell dives into Hyponatremia — a deceptively simple topic that’s a PYQ favorite in NEET PG. Learn how to decode symptoms, spot key lab clues, and master the emergency treatment protocols. With mnemonics, clinical pearls, and exam hacks, this episode is your shortcut to scoring high and saving lives.
What You’ll Learn:
- Types of Hyponatremia: Hypovolemic, Euvolemic, Hypervolemic
- Key causes: SIADH, diuretics, heart failure
- Danger signs: Seizures, altered sensorium
- Treatment essentials: Hypertonic saline, fluid restriction
- Mnemonic mastery: “SHOCKED” for severe signs
🎯 NEET PG Motto:
"PYQs aren’t just questions — they’re clues to what matters most. Every episode of Dr. StemCell Podcast is designed to turn past questions into future marks."
#NEETPG #NEETPG2026 #NEETPGPreparation #NEETPGExam #NEETPGSyllabus #NEETPGStudyPlan #NEETPGCoaching #NEETPGTopperTips #HowToCrackNEETPG #NEETPGQuestionPaper #NEETPGMockTest #NEETPGStrategy #NEETPGTips #NEETPGPreviousYearQuestions #NEETPGOnlineCoaching #NEETPGDailyRoutine #NEETPGNotes #NEETPGImportantTopics #NEETPGStudyMaterial #NEETPGExamPattern #BestBooksForNEETPG #MBBS #MedicalStudents #MedicalEducation #MedSchool #MedStudy #Anatomy #Physiology #Biochemistry #Pathology #Pharmacology #Microbiology #Medicine #Surgery #OBGYN #Pediatrics #Radiology #Orthopedics #Dermatology #Psychiatry #Anesthesia #ENT #Ophthalmology #EmergencyMedicine #CommunityMedicine #INICET #FMGE #AIIMS #JIPMER #PGEntrance #MedicalEntrance
🎧 Episode Title: Hyperkalemia — High-Yield NEET PG Quick Shot
Description:
In this rapid-fire NEET PG prep episode, Dr. StemCell breaks down Hyperkalemia — a small topic with massive exam impact. Learn the must-know ECG changes, emergency treatments, and high-yield mnemonics like TWNS and C BIG K DROP. Avoid common mistakes, master the clinical clues, and lock in your marks with this 4-minute power-packed review.
Key Takeaways:
- ECG signs: Tall T, Wide QRS, No P, Sine waves
- First-line emergency: IV Calcium gluconate
- Mnemonic mastery: C BIG K DROP for treatment steps
Subscribe and follow Dr. StemCell on Apple Podcasts, Spotify, Instagram, and more. Study smart, stay sharp!
#NEETPG #NEETPG2026 #NEETPGPreparation #NEETPGExam #NEETPGSyllabus #NEETPGStudyPlan #NEETPGCoaching #NEETPGTopperTips #HowToCrackNEETPG #NEETPGQuestionPaper #NEETPGMockTest #NEETPGStrategy #NEETPGTips #NEETPGPreviousYearQuestions #NEETPGOnlineCoaching #NEETPGDailyRoutine #NEETPGNotes #NEETPGImportantTopics #NEETPGStudyMaterial #NEETPGExamPattern #BestBooksForNEETPG #MBBS #MedicalStudents #MedicalEducation #MedSchool #MedStudy #Anatomy #Physiology #Biochemistry #Pathology #Pharmacology #Microbiology #Medicine #Surgery #OBGYN #Pediatrics #Radiology #Orthopedics #Dermatology #Psychiatry #Anesthesia #ENT #Ophthalmology #EmergencyMedicine #CommunityMedicine #INICET #FMGE #AIIMS #JIPMER #PGEntrance #MedicalEntrance
Dr. Stem Cell Podcast Episode is Ready!
I've created a comprehensive NEET PG preparation podcast episode that includes:
📚 Content Covered:
Cardiovascular Physiology - Frank-Starling mechanism with
#NEETPG #NEETPG2026 #NEETPGPreparation #NEETPGExam #NEETPGSyllabus #NEETPGStudyPlan #NEETPGCoaching #NEETPGTopperTips #HowToCrackNEETPG #NEETPGQuestionPaper #NEETPGMockTest #NEETPGStrategy #NEETPGTips #NEETPGPreviousYearQuestions #NEETPGOnlineCoaching #NEETPGDailyRoutine #NEETPGNotes #NEETPGImportantTopics #NEETPGStudyMaterial #NEETPGExamPattern #BestBooksForNEETPG #MBBS #MedicalStudents #MedicalEducation #MedSchool #MedStudy #Anatomy #Physiology #Biochemistry #Pathology #Pharmacology #Microbiology #Medicine #Surgery #OBGYN #Pediatrics #Radiology #Orthopedics #Dermatology #Psychiatry #Anesthesia #ENT #Ophthalmology #EmergencyMedicine #CommunityMedicine #INICET #FMGE #AIIMS #JIPMER #PGEntrance #MedicalEntrance
NEET PG, NEET PG preparation, NEET PG 2026, NEET PG exam, NEET PG syllabus, NEET PG study plan, NEET PG coaching, NEET PG topper tips, How to crack NEET PG, NEET PG question paper, NEET PG mock test, NEET PG exam strategy, NEET PG preparation tips, NEET PG previous year questions, NEET PG online coaching, NEET PG daily routine, NEET PG notes, NEET PG important topics, NEET PG study material, NEET PG exam pattern, Best books for NEET PG, MBBS NEET PG preparation, PG medical entrance exam, NEET PG tips and tricks, NEET PG preparation motivation, NEET PG video lecture, NEET PG revision tips, NEET PG flashcards, NEET PG concepts, NEET PG high yield topics, NEET PG clinical subjects, NEET PG anesthesia topics, NEET PG radiology topics, NEET PG ENT topics, Medical entrance exam India
Welcome back to NEET PG 2026 Rapid Round. Today, we dive into hyponatremia — a crucial emergency topic frequently tested in previous years. We’ll cover how to quickly triage, spot causes, and safely correct sodium levels, while avoiding common exam traps.
First, confirm true hypotonic hyponatremia by checking serum osmolality. Remember, hypotonic means osmolality under 275 mOsm/kg. Rule out pseudohyponatremia caused by things like high lipids or proteins, and translocational hyponatremia from hyperglycemia.
Next, assess symptom severity. Severe signs include seizures, coma, or brain herniation threats. Moderate symptoms might be vomiting, confusion, or severe headache. Mild or no symptoms need less aggressive management.
To pinpoint the underlying cause, check the patient’s volume status. Hypovolemia often comes from vomiting or diuretics. Euvolemia can suggest SIADH, hypothyroidism, or adrenal insufficiency. Hypervolemia points to heart failure, liver disease, or nephrotic syndrome.
Treatment depends on these findings. Severe symptoms get quick 3% hypertonic saline boluses to raise sodium safely by 4 to 6 mEq/L initially. Hypovolemic cases respond to isotonic saline. SIADH takes fluid restriction and sometimes salt or loop diuretics. Hypervolemic states need fluid and salt restriction plus diuretics.
Know these key numbers—never exceed an 8 to 10 mEq/L sodium rise in 24 hours to avoid osmotic demyelination syndrome. Many sources suggest aiming for 4 to 6 mEq/L first day for chronic cases.
Let’s test this with some rapid MCQs.
Question one: A 65-year-old on thiazides has confusion with sodium 112, low osmolality, and urine sodium 50. What’s the best first step?
The correct answer: give a 3% sodium chloride bolus to quickly correct the severe symptomatic hyponatremia before addressing diuretics.
Question two: A young adult with pneumonia has sodium 122, euvolemic, concentrated urine with high sodium. Best next step?
Fluid restriction is the right move for SIADH here, not saline infusion, which may worsen dilution.
Question three: What combination increases osmotic demyelination risk?
The answer: chronic low sodium plus malnutrition and correction faster than 10 mEq/L in 24 hours.
If sodium rises too fast, the safest fix is to start D5W and desmopressin to slow down correction and prevent brain damage.
One more: Hyperglycemia with sodium 124 and glucose 600 mg/dL. True or false: This is pseudohyponatremia and needs no correction?
Actually, the sodium needs correcting upward for glucose level before treating sodium abnormalities.
Why use 3% saline boluses in emergencies? Boluses act quickly and can be precisely titrated to the needed 4–6 mEq/L rise without overshooting.
Why avoid normal saline in SIADH? Because kidneys keep the sodium and retain water, worsening hyponatremia with saline.
In persistent euvolemia, always check thyroid and adrenal function before diagnosing SIADH.
Remember potassium too — low potassium correction can raise sodium and cause overcorrection risk.
To wrap up, here’s a quick case: A middle-aged man with lung cancer develops confusion and hyponatremia with high urine sodium and osmolality indicating SIADH. Mild symptoms call for fluid restriction and salt tablets, while severe symptoms deserve 3% saline boluses.
The take-home mantra: Hypotonic first, check volume next; bolus when bad; 4–6 initial correction; never over 8–10; use desmopressin if sodium rises too fast.
That’s all for today’s session on hyponatremia. Stay sharp, stay safe, and control those corrections! Next episode, we’ll quickly cover hyperkalemia ECG patterns and emergency management.
Thank you for listening!
🎙 Spotify Episode Description (copy-paste ready)
Episode Title:
🔑 Pediatric Rickets Simplified — Types, Features, Mnemonics & PYQs
Episode Description (for Spotify):
Rickets keeps coming back in NEET-PG & INI-CET — both in short stems and clinical vignettes. In this episode, we break it down into bite-sized, exam-friendly pearls with easy mnemonics:
📌 What You’ll Learn:
1. Definition & Basics
• Rickets = defective mineralization of growth plate (children).
• Osteomalacia = defective mineralization of osteoid (adults).
2. Types of Rickets
• Nutritional (Vit D deficiency) → most common.
• Vitamin D–dependent (Type I & II)
• Vitamin D–resistant (Hereditary hypophosphatemic rickets)
• Renal rickets (CKD, renal tubular acidosis).
3. Clinical Features (Mnemonic: RICKETS)
• Rosary (rachitic rosary at costochondral junction)
• Inward bowing of legs (genu varum/valgum)
• Craniotabes + delayed closure of fontanelle
• Kyphoscoliosis & knock knees
• Epiphyseal widening + cupping/fraying of metaphysis
• Tetany (hypocalcemia)
• Stomach protrusion (potbelly, Harrison’s sulcus)
4. Radiology Mnemonic: WFR
• Widened growth plates
• Fraying of metaphysis
• Rachitic rosary
5. Important Lab Patterns
• Nutritional Rickets: ↓Ca, ↓PO₄, ↑ALP, ↑PTH
• Vit D–dependent I: ↓1α-hydroxylase → low calcitriol
• Vit D–dependent II: End-organ resistance → high calcitriol
• Hypophosphatemic: Low phosphate, normal calcium
6. Memory Pegs for Types:
• Type I: Hydroxylase defect — think “I can’t Hydroxylate.”
• Type II: Receptor defect — think “II = Resistant.”
7. PYQ Connections
• NEET-PG 2021: child with frontal bossing + rachitic rosary → diagnosis?
• INI-CET 2022: lab values with low phosphate, normal calcium → hypophosphatemic rickets.
• NEET-PG 2018: child with multiple fractures, vit D supplementation not working → Vit D-dependent Type II.
🧠 Key Takeaway:
“One table (Ca, PO₄, ALP, PTH) + one mnemonic (RICKETS) = 3–4 sure-shot questions.”
👉 Share this with your study group — one episode can fetch you multiple marks!
🎙️ Dr. Robbins Rascals NeuroQuickReels
Follow & subscribe on Instagram @Dr.RobbinsRascals
MCQ 1
“A 25-year-old has loss of pain/temp on the left from T6 down; fine touch intact. Lesion is?”
A. Left dorsal column T6
B. Right lateral spinothalamic T6
C. Left anterior spinothalamic T6
D. Right dorsal horn T6
Answer: B. Right lateral spinothalamic T6
Pitfall: Don’t swap sides—spinothalamic crosses early, dorsal columns cross in medulla.
MCQ 2
“In early syringomyelia, the first sensory deficit is:”
A. Ipsilateral vibration below lesion
B. Bilateral cape-like loss of pain/temp
C. Contralateral crude touch below lesion
D. Ipsilateral fine touch at lesion level
Answer: B. Bilateral cape-like loss of pain/temp
Pitfall: Avoid confusing segmental motor signs with initial sensory dissociation.
🔔 Don’t miss more NEET PG nuggets—listen on Spotify and follow @Dr.RobbinsRascals!
Segment 1: Question & Answer Deep Dive
Q1 (NEET PG 2024, Shift 2):
“A 30-year-old male presents with burning pain and temperature loss on the right side of his body below T8. Proprioception and fine touch are intact. Where is the lesion located?”
A. Left dorsal column at T8
B. Left lateral spinothalamic tract at T8
C. Right lateral spinothalamic tract at T8
D. Right dorsal horn at T8
Answer: B. Left lateral spinothalamic tract at T8
Explanation:Pain and temperature fibers cross within one or two segments of entry, so a right‐sided sensory loss below T8 indicates a lesion of the contralateral lateral spinothalamic tract. Proprioception spared because dorsal columns ascend ipsilaterally.
Pitfall to Avoid: Don’t reverse sides! Always map decussation: spinothalamic crosses early; dorsal columns cross in the medulla.
Q2 (NEET PG 2023):
“In syringomyelia, the most characteristic sensory loss pattern is:”
A. Ipsilateral loss of pain and temperature in a cape-like distribution
B. Ipsilateral loss of fine touch and vibration below lesion
C. Contralateral loss of pain and temperature below lesion
D. Ipsilateral LMN signs at level of lesion
Answer: A. Ipsilateral loss of pain and temperature in a cape-like distribution
Explanation:
Central canal expansion disrupts decussating spinothalamic fibers at that
segment, producing a bilateral “cape-like” loss over shoulders and arms. Motor signs may appear later but not the initial finding.
Pitfall to Avoid: Don’t confuse segmental LMN signs (ventral horn) with early sensory deficits—PYQs focus on sensory dissociation first.
Segment 2: Rollouts & Dangers
When tackling spinothalamic MCQs, remember:
1. Rollout Strategy:
Identify modality (pain/temp vs. touch/pressure).
Locate decussation (spinal cord vs. medulla).
Map side of deficit to lesion side (same for touch, opposite for pain).
2. Common Dangers:
Option Overlap: Distractors often mix up tract names (anterior vs.
lateral).
Level Traps: Lesions above T6 but deficits described below T6 – always
check dermatome maps!
Symmetry Tricks: Questions may phrase bilateral vs. unilateral—watch
adjectives like “cape-like” or “stocking-glove.”
Segment 3: Rapid-Fire MCQ Drill
I’ll read five true/false statements—say “true” or “false” out loud!
1. Lateral spinothalamic tract carries crude touch.
2. Pain and temperature cross at the anterior white commissure.
3. Anterolateral system = spinothalamic + spinoreticular tracts.
4. Syringomyelia first affects lower limb pain sensation.
5. Fine touch decussates in the brainstem.
Ready? Pause the podcast, decide, then play back for answers…
[3-second pause]
1 False (crude touch = anterior spinothalamic)
2 True
3 True
4 False (cape-like upper trunk)
5 True
Segment 4: Mnemonic Corner
Mnemonic:
“Love Pain Today”
Lateral = Pain/Temp
Anterior = Touch
Repeat it when you see “spinothalamic” in an MCQ option!
summary of your podcast episode on **Spinothalamic Tracts for NEET PG MCQs**—crafted like something you might hear while tuning in 🎙️:
---
**🎧 NEET PG Neuro QuickPod: Spinothalamic Tract Essentials**
_"Welcome back, future doctors! In today’s brain-hugging episode, we’re diving into a super high-yield topic—Spinothalamic tracts. This is one of those core neuro pathways that pops up across PYQs and is a must-know for NEET PG MCQs. So let’s break it down, mnemonic-style and clinically sharp!"_
🧠 **Segment 1: Quick Anatomy Refresher**
- The spinothalamic tract carries **pain, temperature, crude touch, and pressure** sensations.
- It consists of two parts: **anterior spinothalamic tract (touch & pressure)** and **lateral spinothalamic tract (pain & temperature)**.
- Originates from dorsal horn neurons → crosses to the opposite side at the spinal level → ascends to the thalamus → projects to somatosensory cortex.
📌 **NEET PG Alert: MCQ Trap Watch**
- **Lesion localization**: A right-sided spinal cord lesion affects the **left-sided pain/temp** due to decussation.
- Common question: _"Loss of pain and temperature on left below T10. Where’s the lesion?"_
🧪 **PYQ Highlights**
- NEET PG 2022: Asked about loss of pain/temp below lesion level—correct answer involved lateral spinothalamic tract.
- NEET PG 2019: Image-based MCQ—match sensory pathway with lost modality.
🧬 **Clinical Correlations**
- Conditions like **syringomyelia** (central canal cyst) first affect spinothalamic tract fibers → leads to dissociated sensory loss.
- A favorite examiner trick: showing bilateral loss of pain/temp in a cape-like distribution.
💡 **Mnemonic Corner**
_"Pain and Temp cross early, touch and pressure take their time."_
Think: **'Lateral Love Pain'** (Lateral spinothalamic = pain/temp), **‘Anterior Affection Touch’** (Anterior = touch/pressure).
✅ **MCQ Strategy**
- Track decussation points.
- Don’t mix up dorsal column vs spinothalamic pathways (watch for distractors).
- Look for patterns of sensory loss vs motor loss.
_"That’s all for today’s neuro nugget. Remember, repetition is the key—and now you’re spinothalamic-savvy! Keep smashing those MCQs and we’ll catch you in the next mini-review!"_
DIFFERENT MEDICAL CASES WE HAVE DISCUSSED GREAT TO LISTEN FOR NEET PG
FEW QUESTIONS OF OBGY IN NEET PG
HERE IS A SMALL DISCUSSION OF 27 questions of neet pg 2024 shift 2 questions in 10 minutes
THIS PODCAST IS ABOUT THE NEET PG 2024 question paper shift 1 ,
rapid questions and answers in one line as a discussion in 7 min of the whole
This 22-minute episode covers key concepts in Anatomy & Embryology for NEET PG, INICET, and FMGE. From germ layers and pharyngeal arches to diaphragmatic defects, cranial nerves, spinal tracts, heart development, and limb innervation—Dr. Stem Cell breaks it all down with clinical relevance, mnemonics, and exam tips. A must-listen rapid revision for serious med aspirants!